laryngomalacia is the most common cause of chronic stridor in infants, resulting from the structural immaturity of the laryngeal cartilages. During inspiration, the epiglottis and/or arytenoid cartilages are pulled into the airway lumen due to negative intrathoracic pressure, creating a partial obstruction. While often self-limiting and resolving by 18–24 months, severe cases may require surgical intervention. It is distinct from subglottic stenosis (Q31.1) or vocal cord paralysis (J38.0), which present with different acoustic signatures. Clinical subtypes include anterior epiglottic collapse, arytenoid prolapse, and short aryepiglottic folds, each coded under Q31.5.
The term entered medical English in the late 19th century to describe the “softening of the larynx.” It combines the anatomical root for the voice box with the pathological suffix for tissue softening, mirroring terms like osteomalacia (softening of bone).
🔀 ALIASES / ALTERNATE TERMS
Laryngomalacic(adjectival descriptor for airway findings)
⚠️ Coding Note: When coding for Q31.5, ensure the documentation specifies the severity and the presence of failure to thrive or apnea, as these may support higher-level E/M codes. supraglottoplasty (31546) is the definitive surgical procedure; ensure the operative report clearly describes the division of the aryepiglottic folds. If the patient has comorbid GERD, code K21.9 as a secondary diagnosis to justify medical management (e.g., PPI therapy).